Advances in Virus Research, Volume 71 by Karl Maramorosch

By Karl Maramorosch

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And Wimmer, E. (1995). Mouse neuropathogenic poliovirus strains cause damage in the central nervous system distinct from poliomyelitis. Microb. Pathog. 18:253–267. , and Wimmer, E. (1996). Internal ribosomal entry site substitution eliminates neurovirulence in intergeneric poliovirus recombinants. Proc. Natl. Acad. Sci. 93:2370–2375. , Bowman, V. , Bator, C. , Baker, T. , Kuhn, R. , and Rossmann, M. G. (1999). Interaction of the poliovirus receptor with poliovirus. Proc. Natl. Acad. Sci. 97:79–84.

Important determinants in the 50 noncoding region are located at sites involved in the secondary structure of RNA, which may influence the initiation of translation at the internal ribosome entry site (IRES). , 1996). A virulent poliovirus was compared with a recombinant bearing the IRES from human rhinovirus type 2. Both viruses grew equally well in HeLa cells but the recombinant virus scarcely replicated in human neuroblastoma cells and failed to paralyze PVR transgenic mice, while the virulent virus replicated well in neuroblastoma cells and had a high intracerebral LD50 titer in transgenic mice.

J. Virol. 61:2917–2920. , and Popper, E. (1909). Ubertragung der Poliomyelitis acuta auf Affen. Z. Immunitatstorsch. Orig. 2:377–390. Lennette, E. , and Schmidt, N. J. (1957). Studies on the development and persistence of complement-fixing and neutralizing antibodies in human poliomyelitis. Am. J. Hyg. 65:210–238. Leon-Monzan, M. , and Dalakas, M. C. (1995). Detection of poliovirus antibodies and poliovirus genome in patients with the post-polio syndrome. Ann. N. Y. Acad. Sci. 753:208–218. Macadam, A.

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